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Therefore, this lesion, in the absence of detectable morphologic cause for its development, could be construed to be a lymphangioma, similar to the lesions reported ten years ago in three boys aged 13, 16, and 19 years. In two of those cases, there was associated mediastinal involvement, and two lobes were affected. On the other hand, one cannot easily dismiss the possible role played by pulmonary scarring which may have developed following the stormy perinatal period this child went through, and which required mechanical ventilation for the first three months of life. Lymphangiectasis does develop in the course of bronchopulmonary dysplasia; and although at nine years of age, there is no evidence of residual pulmonary damage, it is not inconceivable that this might have played a role in the pathogenesis of the present lesion. Unfortunately, at this stage, we know of no way to answer the question, although a significant future increase in the number of cases of late-onset localized pulmonary lymphatic anomalies would definitely favor such an explanation. In the lungs, neonatal lymphangiectasis is perceived as resulting from diffuse dysplasia of the lymphatic network. read only
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